Thoracic Key

Abstract A detailed understanding of the anatomy of the atrial septum and its relationship with critical structures such as the aortic root and the posterior atrial wall is crucial to…

Abstract Idiopathic ventricular tachycardias (VTs) usually occur in specific locations and have specific QRS morphologies. The most common idiopathic VT is VT from the outflow tract of the right ventricle…

Abstract Ventricular tachycardia (VT) in the setting of structural heart disease, whether ischemic or nonischemic in etiology, is usually caused by reentry caused by the presence of scar. Catheter ablation…

Abstract Children are usually smaller than adults, but in general ablation techniques used in adults should not simply be miniaturized to fit the size of the pediatric patient. Multiple factors,…

Abstract The ability to map the epicardial surface of the heart has led to significant advances in our understanding of arrhythmogenic substrates associated with disease-specific etiologies. Access to the pericardial…

Abstract Ventricular tachycardia (VT) can develop late after myocardial infarction, often in the context of advanced systolic dysfunction, potentially resulting in hemodynamic instability and sudden death. Although implantable cardioverter-defibrillators are…

Abstract A body of work has developed to supplement the classic methods of activation and entrainment mapping for cases of hemodynamically unstable ventricular tachycardia (VT). Definition of the arrhythmic substrate…

Abstract Monomorphic ventricular tachycardia in postoperative congenital heart disease patients is based upon morphologic/anatomic variants of the heart defect itself or caused by ventricular incisions and patches that allow initiation…

Abstract Ventricular tachycardia (VT) is an outcome-defining complication in the natural history of non-ischemic cardiomyopathies, including idiopathic dilated cardiomyopathy, cardiac sarcoidosis, arrhythmogenic right ventricular cardiomyopathy, among others. While a number…

Abstract In many cases, hemodynamic instability limits the ability to perform activation mapping during scar-related ventricular tachycardia (VT). Catecholamines may support blood pressure, but overall perfusion may be suboptimal, and…