Syncope

CHAPTER


15



Syncope


UNDERSTANDING AND MANAGING SYNCOPE


Syncope is defined as a sudden, transient loss of consciousness and postural tone with spontaneous recovery.


Incidence of syncope is 6.2/1000 person years with a 30% recurrence.


Syncope accounts for 3% of emergency room (ER) visits and 1%–6% of hospital admissions.


If the cause of syncope is cardiac, there is a 20% 1-year mortality rate.


Etiology


Cardiac causes (20%)


Arrhythmia


Tachyarrhythmia: Supraventricular tachycardia (SVT), atrial fibrillation (AF)/atrial flutter (AFL), ventricular tachycardia (VT)


Bradyarrhythmia: Bradycardia, AV block, or bundle branch block (BBB)


Mechanical obstruction


Valvular: Aortic stenosis, pulmonary stenosis, mitral stenosis


Hypertrophic obstructive cardiomyopathy


Pump failure: MI, tamponade


Other: Atrial myxoma, pulmonary embolism


Neurocardiogenic causes (60%)


Pathophysiology (Bezold-Jarisch reflex)


Peripheral venous pooling results in a drop in preload triggering a hypercontractile state. This results in mechanoreceptor stimulation (posterior-inferior LV) triggering a medullary vasodepressor brain stem reflex. This results in decreased sympathetic and increased vagal tone, leading to lower heart rate (HR), blood pressure (BP), and blood vessel tone.


Subtypes:


Carotid sinus hypersensitivity (lower BP, lower HR, or both)


Vasovagal syncope: “Common faint,” cardioinhibitory, and vasodepressor variants


Situational syncope: Micturition, defecation, deglutition, cough, swallow, pain


Neurologic


Vertebrobasilar insufficiency, usually associated with dysarthria and dysphagia


Subarachnoid haemorrhage


Seizure (bicortical failure): Sudden loss of postural tone followed by tonic-clonic movements. Incontinence and post-event confusion are common.


Atypical migraine


Orthostatic hypotension


Pathophysiology: Postural hypotension resulting in symptoms of cerebral hypoperfusion, leading to decreased effective circulating volume


Potential causes include:


Volume depletion due to hemorrhage, diuresis, diarrhea, sepsis, salt wasting, and supine hypertension


Hyperthyroid, diabetes (hyperglycemia), pheochromocytoma, adrenal insufficiency, diabetes insipidus (DI)


Venous pooling: Postural, morning, postprandial, pregnancy


Drugs


Antihypertensive (sympatholytic, vasodilator, diuretic), nitrates, alcohol


Antidepressant (tricyclic antidepressant [TCA]), antipsychotic, anti-Parkinsonian drugs, benzodiazepines, narcotics


Autonomic neuropathy (diabetes mellitus, alcohol, B12, amyloid, Bradbury-Eggleston, Shy-Drager)


Neurologic (stroke, posterior fossa tumours, upper cervical cord lesions)


Other


Psychiatric: Somatization, pain, anxiety


Metabolic: Hypoglycemia, dumping syndrome, hypoxia, hypocapnea


Clinical


History/physical examination is diagnostic in 40%–50% (50% of cases are undiagnosed).


Structured history may help improve the diagnostic yield, and is evidence-based:


Calgary syncope score (Eur Heart J. 2006;3:344–350); vasovagal syncope if score ≥ –2; see Table 15.1


Table 15.1 Calgary Syncope Score






























Component Score

1. History of bifascicular block, asystole, SVT, diabetes


–5


2. History of bystanders noting a bluish skin tone during the episode


–4


3. Syncope beginning after the age of 35 years


–3


4. Do you remember anything about being unconscious


–2


5. Lightheaded spells or fainting with prolonged sitting or standing


  1


6. Do you sweat or feel warm before a faint?


  2


7. Do you have lightheaded spells or faint with pain or in medical settings?


  3


Age of onset


Young


Predominantly these cases are vasovagal.


Uncommon causes such as idiopathic VT, prolonged QT, and HCM should be considered with an atypical history.


Older


Strongly consider an arrhythmic cause.


Number of episodes


Single/multiple over long period (~benign)


Multiple over short period (~malignant)


Table 15.2 Characteristics of Syncope Episodes that Point to Neurocardiogenic vs. Arrhythmia































Neurocardiogenic Arrhythmia

Triggers


Prolonged standing


Pain, emotional upset, fear/anxiety


CSH: Turning head, seat belt, shaving


Situation: Defecation, coughing, eating, drinking


Seated or supine


Prodrome


Nausea, diaphoresis, presyncope


Pallor, palpitations, visual changes


Sudden LOC without warning


Onset – Offset


Abrupt – Slow


Abrupt – Abrupt


Resolution


Immediately after lying down


Residual symptoms


Fatigue/weakness (usually severe), nausea


Diaphoresis, headache


CSH: carotid sinus hypersensitivity; LOC: loss of consciousness.


Medical history


Diabetes


Psychiatric disorders


Coronary arterial disease (CAD) (if present, then 26% chance cardiac syncope; if absent, then 24% vasovagal syncope)


Medications/drugs (especially changes):

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Feb 28, 2017 | Posted by in CARDIOLOGY | Comments Off on Syncope

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