Objective
Sympathetic overactivity plays a critical role in the development, maintenance, and aggravation of ventricular tachyarrhythmia (VT). We aimed to report our case series with 3-month outcomes in patients with implantable cardioverter-defibrillators (ICD) undergoing renal artery sympathetic denervation (RASD) for refractory VT.
Methods
A total of 4 patients with refractory VT underwent RASD. Underlying conditions were ischemic dilated cardiomyopathy (n = 3), and non-ischemic cardiomyopathy (n = 1). Information on the number of VT episodes and device therapies (anti-tachycardia pacing/shocks) in the previous 3 months as well as 3 months after RASD treatment was obtained from ICD interrogation. Renal artery denervation was incomplete in 1 patient. In one patient both renal arteries and accessory left renal artery were denervated.