The report by Stöllberger et al in the October 1, 2011, issue of The American Journal of Cardiology on the frequency of stroke and embolism in 144 patients with left ventricular hypertrabeculation/noncompaction, a cardiac abnormality of unknown origin, was based on a retrograde analysis of baseline clinical, echocardiographic, and electrocardiographic data. In reference to electrocardiographic information, the investigators reported on the prevalence of normal findings, left bundle branch block, pathologic Q waves, and atrial fibrillation. Left ventricular hypertrabeculation/noncompaction is characterized by trabeculations in the inner core of myocardium and a thinner than usual external compact myocardial core. One wonders, given these anatomic peculiarities, about possible changes in the depth distribution and/or extent of the Purkinje ventricular conduction network in patients with left ventricular hypertrabeculation/noncompaction. Is it possible that intraventricular conduction is altered in such patients? I will be grateful to the investigators if they provide data on the electrocardiographic QRS durations, QT and corrected QT intervals, and PR intervals of their study patients.