3.13 Ventricular Tachycardia Slow Monomorphic Ventricular Tachycardia Following Anterior Infarction, Spontaneous Termination Etiology: Coronary heart disease with status post infarct Mechanism: Area in the region of the infarction scar with delayed impulse conduction facilitates a reentry circuit ECG characteristics: Tachycardia with wide QRS complex Acute treatment: Defibrillation If hemodynamically stable, also attempt termination with antiarrhythmics (class IB, amiodarone) in intensive care Chronic treatment: In the majority of cases implantation of a defibrillator (ICD) Antiarrhythmics and ablation treatment rarely curative, more frequently used as adjuvant treatment to reduce the frequency of arrhythmic episodes Idiopathic Ventricular Tachycardia Idiopathic ventricular tachycardia arising from the right ventricular outflow tract (RVOT) Mechanism: Triggered activity or abnormal automation ECG characteristics: Tachycardia with wide QRS complex Inferior axis (ranging from right to marked right axis deviation); left bundle branch configuration V1: “QS” configuration (absent “r“) Tall narrow “R” in leads II and III Small “R” or “rS” in lead I Repetitive monomorphic right ventricular tachycardia (GALLAVERDIN) Mechanism: Triggered activity ECG characteristics: Tachycardia with wide QRS complex Left bundle branch configuration Inferior axis (right to marked right deviation) Only gold members can continue reading. Log In or Register to continue Share this:Click to share on Twitter (Opens in new window)Click to share on Facebook (Opens in new window) Related Related posts: The Isthmus Region Acute Coronary Syndrome Resting Ischemia in the Anterior Wall Region Following Posterior Wall Infarction Medication-Related ECG Changes Stay updated, free articles. Join our Telegram channel Join Tags: Easy ECG Aug 29, 2016 | Posted by admin in CARDIOLOGY | Comments Off on Ventricular Tachycardia Full access? Get Clinical Tree
3.13 Ventricular Tachycardia Slow Monomorphic Ventricular Tachycardia Following Anterior Infarction, Spontaneous Termination Etiology: Coronary heart disease with status post infarct Mechanism: Area in the region of the infarction scar with delayed impulse conduction facilitates a reentry circuit ECG characteristics: Tachycardia with wide QRS complex Acute treatment: Defibrillation If hemodynamically stable, also attempt termination with antiarrhythmics (class IB, amiodarone) in intensive care Chronic treatment: In the majority of cases implantation of a defibrillator (ICD) Antiarrhythmics and ablation treatment rarely curative, more frequently used as adjuvant treatment to reduce the frequency of arrhythmic episodes Idiopathic Ventricular Tachycardia Idiopathic ventricular tachycardia arising from the right ventricular outflow tract (RVOT) Mechanism: Triggered activity or abnormal automation ECG characteristics: Tachycardia with wide QRS complex Inferior axis (ranging from right to marked right axis deviation); left bundle branch configuration V1: “QS” configuration (absent “r“) Tall narrow “R” in leads II and III Small “R” or “rS” in lead I Repetitive monomorphic right ventricular tachycardia (GALLAVERDIN) Mechanism: Triggered activity ECG characteristics: Tachycardia with wide QRS complex Left bundle branch configuration Inferior axis (right to marked right deviation) Only gold members can continue reading. Log In or Register to continue Share this:Click to share on Twitter (Opens in new window)Click to share on Facebook (Opens in new window) Related Related posts: The Isthmus Region Acute Coronary Syndrome Resting Ischemia in the Anterior Wall Region Following Posterior Wall Infarction Medication-Related ECG Changes Stay updated, free articles. Join our Telegram channel Join