Liver cirrhosis, consequence of a nodular regenerative hyperplasia, complicated by hypersplenism with thrombopenia (platelets 28,000 G/l), and bleeding esophageal varices. A restoration of the platelet count is researched. (a) CT frontal reconstruction: splenomegaly. (b) Splenomegaly, large splenic vein. (c) Large diameters of portal vein and its branches. (d) Celiac trunk (CT) selective angiogram: large and sinuous splenic artery. (e) Distal splenic A hyperselective catheterization (microcatheter): modal pattern in bifurcation; microaneurysms. (f) Hyperselective catheterization of the lower terminal branch, which has been occluded with microparticles, and then catheterization and occlusion of the lower branch of the superior terminal branch. At the end of this procedure, 4 cm³ of 300–500 μm diameter particles, and then 3 cm³ of 500–700 μm diameter, and finally 1 cm³ of 900–1,200 μm diameter have been injected, in order to observe a considerable slowing of the flow in these branches. (g) The upper pole of the spleen has not been embolized. (h) CT, 1 month later: 60–70 % devascularization of the splenic parenchyma. During the immediate follow-up: moderate fever (38 °C), limited pleural effusion, and platelet count progressively arising (from 28 × 10³ to 109 × 10³ G/l) without any pancreatic enzymes movement