Background
TRC inducing trauma to radial artery intima may induce anatomical and functional changes at late term. However, there is paucity of data about the changes in radial artery diameter and endothelial functions just before discharge following the exclusion of cases with acute radial artery thrombotic occlusion.
Background
TRC inducing trauma to radial artery intima may induce anatomical and functional changes at late term. However, there is paucity of data about the changes in radial artery diameter and endothelial functions just before discharge following the exclusion of cases with acute radial artery thrombotic occlusion.
Methods
Fifty eight consecutive patients undergoing TRC were enrolled prospectively. Baseline RAD, flow mediated dilatation (FMD) and nitroglycerin mediated dilatation (NMD) of the radial artery at the access site were measured before TRC by using high resolution ultrasound. Terumo band compression device was used with reverse Barbeau test to achive patent hemostasis after TRC. RAD, FMD and NMD were re-measured just before discharge after hemostasis of the radial artery access site. Patients with acute occlusion of the radial arteries were excluded.
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