Introduction
Abdominal aorta aneurysm (AAA) is a progressive vessel disease which happens when the aorta loses its normal structure. The aortic wall gets thinner and the diameter expands more than 1.5-2 times of its normal diameter. It’s the thirteenth most diagnosed reason of death in society and the prevelance is higher in males. In cases with diameter of the aneurysm is more than 8 cm, mortality rate is %30-50. In elective surgery, mortality rate is less than %5, in emergency surgery the rate is %25 and in ruptured aneurysm is %54. 5 year survival rate is %50 and the rate decreases to %40 in 10 years. 200.000 patients are diagnosed with AAA in USA and 15.000 of them dies per year.
Case Presentation
A 65 year old male patient with ruptured AAA was referred to our clinic from another medical centre for emergency surgery. Computed tomography (CT) scans showed AAA with 10 cm diameter. Physical examinations revealed hypovolemic shock and hypotension. Aorta-bifemoral bypass was performed and 5450 cc blood product was given to patient during the surgery. Peroperative multiple and high dose inotropic agents and transamine infussion treatment was given. Metabolic acidosis and dissemine intravasculer coagulation (DİC) was developed after surgery, hemoglobin value was 4.7g/dl, hematocrit was 14.4, platelet was 38000/uL. Total amount of 2700 cc blood product was given after surgery and the patient bled 2000 cc. Patient was extubated in post operative 5. day and was discharged from intensive care unit in 9. day. Patient was taken back to the intensive care unit due to massive bronchial secretion and poor general health condition. After 14 days of observation in intensive care unit and 12 days in ward, patient was discharged. Total of 11800 cc blood and blood product was given to patient during hospitalisation.
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