Objective

Several studies have shown the association between STEMI and circadian rhythm. Nonetheless, intrinsic circadian release or sequestering of platelets is controlled by various humoral factors, including hormones, endothelial-derived factors, platelet-derived factors and other blood-borne substances. We hypothesized that endogenous circadian rhythm in platelet function may play a partial role in the circadian rhythm of STEMI.

Material-Methods

A total of 205 patients with STEMI were enrolled into this study. Hospital’s computerized database was used to obtain clinical and demographic characteristics of patients. The time in which chest pain had started was considered as the beginning of AMI; within ±30 min. Patients were then categorized into 4 6-h increments (12:00 AM–06:00 AM, 06:00 AM–12:00 PM; 12:00 PM–06:00 PM and 06:00 PM–12:00 AM hours). Basal demographic, laboratory and echocardiographic features of the patients were compared between two groups.

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