This comment is in response to the contribution by Greutmann et al in the January 15, 2012, issue of The American Journal of Cardiology on adverse outcomes in 155 patients with isolated left ventricular (LV) noncompaction, 77% of whom were symptomatic when their condition was identified. At a median follow-up time of 2.7 years, none of the asymptomatic and 31% of the symptomatic patients had died or undergone transplantation. The major determinants of these 2 outcomes were presentation with a cardiovascular complication, New York Heart Association class ≥III. As in a study by Stöllberger et al recently published in the Journal , the electrocardiographic data are scanty. Indeed, the investigators listed in Tables 1, 2, and 4 only atrial fibrillation and bundle branch block (type not specified) and in Table 3 sustained ventricular tachycardia. It would have been appropriate at least to report on LV hypertrophy, considering that their symptomatic patients had higher rates of LV dilatation and decreased LV ejection fractions. The investigators refer to “the thickened 2-layered myocardium consisting of a thin compacted outer (epicardial) and a much thicker noncompacted inner (endocardial) layer with a deep intratrabecular recesses”; is it possible that the intraventricular conduction is affected in patients with LV noncompaction because of alterations in the extent, distribution, or functional characteristics of the Purkinje network or the absolute thickness of the compacted and noncompacted myocardial layers? Stöllberger et al reported on the presence of left bundle branch block and Q waves in their patients. Could Greutmann et al oblige by supplying information on the frequency of LV hypertrophy, Q waves, left bundle branch block, right bundle branch block, and left anterior and posterior hemiblocks, as well as QRS duration, QT interval, corrected QT interval, and PR interval, in their patients?