Background
Plaque rupture does not always cause acute coronary syndrome (ACS), and many silent plaque raptures have been detected by various imaging modalities. We hypothesized that dynamic change in the thrombogenic potential of blood may play an important role for the onset of ACS among the patents with ruptured plaques.
Objectives
To examine the difference in the thrombogenic potential of blood between the patients with ACS and those with stable coronary diseases.