Questions
Figure 26.5 (a) Right bundle branch block? (b) Pre-excitation? (c) Right ventricular hypertrophy? (d) True posterior myocardial infarction?
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Figure 26.13 Which drug(s) might have been appropriately used? (a) Digoxin? (b) Flecainide? (c) Verapamil? (d) Sotalol? (e) Disopyramide
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Figure 26.14 Why might have this patient complained of rapid heart beating preceding his ‘dizzy attacks’?
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Figure 26.23 Which drug might the patient have been taking? (a) Flecainide? (b) Tetracycline? (c) Bisoprolol? (d) Clarithromycin? (e) Digoxin?
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Figure 26.25 What is the arrhythmia? If catheter ablation were to be carried out, where should radiofrequency energy be delivered?
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Figure 26.26 Leads V1 and V5. What is the arrhythmia? (Why is there changing morphology in lead V1?)
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Figure 26.48 This patient had a very poorly functioning sinus node. How could an appropriate increase in heart rate during exercise be achieved?
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Figure 26.60 This patient experienced syncope but had a normal pulmonary artery pressure. What was the cause?
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Figure 26.69 In spite of digoxin 0.125 mg daily with normal renal function and body weight. What should be done?
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Figure 26.78 This patient had paroxysmal atrial fibrillation and good ventricular function and was prescribed flecainide.
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