Abstract
One of the most troublesome complications of percutaneous coronary intervention (PCI) or angiography is retrograde dissection of the artery into the aortic root. We report a case involving the right coronary artery (RCA) which was treated with prompt deployment of stents. Recurrent chest pain and ST segment elevation in V 2–4 mimicked the ECG appearance of acute anterior infarction and prompted concern that the dissection had extended to impair flow in the left coronary artery (LCA). Transoesophageal echocardiography (TOE) demonstrated that the aortic root dissection had been contained and that the LCA was not compromised.
1
Case report
A 53 year old man presented with an acute inferior ST-elevation myocardial infarct ( Fig. 1 ). Coronary angiography showed no significant disease of the left system. The occluded RCA ( Fig. 2 ) was engaged with a 6 F JR4 guide and a guide wire 1
1 Guidant BMW.
passed to the distal vessel. The lesion was dilated with a 3.0 mm balloon 22 3.0 × 20 mm Guidant Cross-Sail to 10 bar.
to 10 bar. The subsequent angiogram disclosed retrograde dissection to the aortic root ( Fig. 3 ). Two bare-metal stents 33 3.5 × 23 mm Multi-Link Vision™ to 10 bar, 3.5 × 24 mm Medtronic AVE S7 to 14 bar.
were deployed across the lesion back to the ostium of the RCA to seal the entry ( Fig. 4 ). He was given heparin, abciximab, metoprolol, aspirin, and clopidogrel.Two hours later he developed recurrent chest pain. His ECG showed that the inferior ST elevation had resolved but displayed new anterior ST elevation to 4 mm in V 2 and V 3 ( Fig. 5 ). We were concerned that the dissection had extended to compromise flow in the LCA but TOE showed that the haematoma remained confined to the aortic root adjacent to the ostium of the RCA and that the LCA was patent ( Fig. 6 ). Left ventricular systolic function was well-preserved with no regional wall motion abnormalities.
We presumed that his recurrent pain and the transient ECG changes in the anterior precordial leads had reflected occlusion of a conus or right ventricular branch of the RCA, the origin of which had been compromised by our intervention. His anterior ECG changes improved and his pain settled promptly with analgesia, β-blockade and ramipril. He was discharged after six days.
Three months later he was admitted to another hospital with diffuse in-stent restenosis which was corrected with two drug-eluting stents.